InflammatoryCommonICD-10 L30.0

Discoid eczema

Nummular eczema ยท nummular dermatitis ยท discoid dermatitis

Discoid eczema is a chronic-relapsing eczematous dermatitis characterised by discrete, coin-shaped pruritic plaques most commonly on the limbs. It affects ~0.1-0.2% of the UK population, peaking in middle-aged and elderly adults. It is a common DDx for tinea, psoriasis, contact dermatitis and mycosis fungoides. It is also a frequent reason for biopsy when refractory or asymmetric, and a known phenotype of EGFRi / BRAFi / ICI cutaneous reactions.

CurrentLast reviewed 16 May 2026
Clinical image of Discoid (nummular) eczema
Discoid (nummular) eczema. Image sourced from DermNet New Zealand. Used under CC BY-NC-ND 4.0. No endorsement implied.

Pathogenesis

  • Multifactorial:
    • Atopic background โ€” overlap with atopic eczema.
    • Skin barrier dysfunction.
    • Staphylococcus aureus over-colonisation.
    • Cold, dry climate; low humidity (winter eczema).
    • Drug-induced: hydrochlorothiazide, methyldopa, gold, retinoids, interferons, EGFRi.
  • Older adults disproportionately affected.

Clinical features

  • Coin-shaped (nummular) erythematous well-demarcated plaques, 1-10 cm diameter.
  • Vesicular / oozing acutely; lichenified chronically.
  • Distribution: limb extensors (commonest), trunk, hands, lower legs.
  • Intense pruritus.
  • Chronic-relapsing course; commonly bilateral and symmetric.
  • Often secondarily impetiginised (S. aureus).

Differentials

  • Tinea corporis โ€” advancing scaling edge with central clearing; KOH+.
  • Psoriasis โ€” silvery scale, sharply demarcated, Auspitz, nail involvement.
  • Atopic eczema โ€” atopic background, flexural distribution.
  • Pityriasis rosea โ€” herald patch, Christmas tree, collarette scale.
  • Mycosis fungoides โ€” chronic, asymmetric, sun-protected; biopsy + TCR.
  • Contact dermatitis โ€” exposure-related.
  • Bowen disease โ€” keratotic, fixed plaque.
  • Sub-acute lupus erythematosus.
  • Drug-induced eczematous eruption (EGFRi, BRAFi, ICI).

Investigations

  • Clinical diagnosis usually adequate.
  • KOH if tinea suspected.
  • Bacterial swab if oozing / pustular (S. aureus colonisation).
  • Skin biopsy in atypical / persistent / asymmetric lesions to exclude MF, psoriasis, Bowen.
  • Patch testing if contact dermatitis suspected (especially asymmetric or related to nickel-belt-buckle or footwear).
  • Drug history (HCTZ, gold, IFN, EGFRi, BRAFi).

Management

  • General:
    • Bland emollients liberally; avoid soap; soap substitute.
    • Humidify environment.
  • Topical:
    • Potent topical corticosteroid (clobetasol propionate, betamethasone valerate) 2-4 weeks; taper.
    • Topical calcineurin inhibitors โ€” steroid-sparing.
    • Coal-tar containing combinations for chronic lichenified plaques.
  • Bacterial superinfection: oral flucloxacillin / erythromycin / clarithromycin 7 days; topical fusidic acid (avoid prolonged courses).
  • Phototherapy: NBUVB if widespread / refractory.
  • Systemic: methotrexate, ciclosporin, azathioprine, mycophenolate if refractory.
  • Dupilumab: emerging evidence in the nummular / discoid eczema phenotype where disease overlaps with moderate-to-severe atopic dermatitis; confirm local specialist access and commissioning route.
  • Counsel on chronic-relapsing course and trigger avoidance.

References

  1. Bonamonte D et al. Nosological position of discoid eczema. Acta Derm Venereol. 2012;92:586-589.
  2. Ozkaya E. Adult-onset atopic dermatitis. J Am Acad Dermatol. 2005;52:579-582.
  3. NICE CKS. Eczema โ€” atopic. London: NICE; accessed 18 May 2026.
  4. Bonamonte D et al. Topical management of nummular dermatitis. Dermatitis. 2008;19:204-208.

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